Validity of haemoglobin A1c and glycoalbumin for an appropriate evaluation of glycaemic control in Japanese diabetic patients with chronic renal failure

occurs in 10–20% of patients following surgical graft repair of AAA. The most probable cause for this is mechanical due to the compression of the ureter against the native iliac artery from the anteriorly placed graft [2]. In a prospective study of 101 patients who underwent aortofemoral and aortoiliac reconstructive surgery, 12% of patients developed mild to moderate hydronephrosis. All patients were asymptomatic, and the obstruction resolved spontaneously in 10 of 11 patients within 3 months of onset. The incidence of delayed hydronephrosis occurring 1 year or more post surgical repair is unknown. This complication may be relatively common after reconstructive vascular surgery as in our patient, especially in association with infected grafts [3]. The mechanism for the development is not fully understood. The most widely held view is that this complication develops secondary to an inflammatory process causing fibrosis [4]. The long time interval is unusual and suggests perhaps a different mode of inflammatory pathways compared to what is commonly seen in early obstructive uropathy associated with surgical graft repair of AAA. More research is needed to elucidate the mechanisms underlying chronic periaortitis [5].